I hate the way you seem to think you can see in the future and ‘know’ about problems before anyone else.
You have no idea what your talking about! Do some research before making useless posts.
Talking about yourself in the 3rd person doesn’t make you come across as interesting, it makes you look foolish.
Please stop spaming this forum with your gibberish, there are people here that want to do some actual research and learn something interesting not troll through usless crap posts like yours.
Dermabrasion does not go anywhere near the follicles. It merely affects the epidermis and doesn’t go as deep as the dermis, beneath which quite deeply, the follicles reside.
» Arava is Leflunomide. That’s one of the short list of Folica’s internal
» EGF-R inhibition drugs in the patents.
Assuming we can obtain this without a prescription, what dosage size in mg are we talking about? And also, do we take it orally or incorporate it into a topical after the wounding phase? Any ideas?
Edit: I began filling the form out at Goldpharma.com to order 30x 20 mg tablets of Arava. Total cost about 152 Euros. But they required to know what ailment I would be taking the medication for. I could say arthiritis but what if they ask further questions? It seems that they WON’T require a prescription from my doctor anyway.
» » Arava is Leflunomide. That’s one of the short list of Folica’s internal
» » EGF-R inhibition drugs in the patents.
»
» Assuming we can obtain this without a prescription, what dosage size in mg
» are we talking about? And also, do we take it orally or incorporate it into
» a topical after the wounding phase? Any ideas?
»
» Edit: I began filling the form out at Goldpharma.com to order 30x 20 mg
» tablets of Arava. Total cost about 152 Euros. But they required to know
» what ailment I would be taking the medication for. I could say arthiritis
» but what if they ask further questions? It seems that they WON’T require a
» prescription from my doctor anyway.
They wont be asking more question. Just fill in whatever the drug is usually for. Worked just fine for me. Good luck.
The normal arthritis dosage is only like 10mg per day
BUT -
They recommend starting off with a “loading dose” of 100mg/day for the first three days. Otherwise it will take a long time to build up enough drug in the system to feel the effects.
They also say it takes a while to get the stuff back OUT of your system when you stop. (Like months, if you’ve been on the stuff for months already.)
I’m thinking it may just be best to slam a couple hundred mg of the stuff in the first couple of days, and then take little or nothing after that for the rest of the timeframe. Then start taking activated charcoal at the end of the Folica timeframe to begin working it back out of the system.
Thanks. I’m still undecided whether to go for this. I’m partial to alcohol and apparently that’s a no-no on this medication. I’m also concerned that due to the drug requiring build-up time in the body, we may miss our ‘embryonic window’ if we take it after wounding. I’m aware that you should take a loading dose for 3 days but will there be enough drug in the body for our purposes. There are enough uncertainties about our homemade version of this procedure without introducing another wildcard. What’s your thoughts on timeframe for the drug?
If I was pretty sure we’d get it right I would be willing to abstain from alcohol for 10 days or so. Only just.
Thinking about it, I think maybe topical application is the way to go. Dissolve tabs in some DMSO and add this to emu oil.
» » Arava is Leflunomide. That’s one of the short list of Folica’s internal
» » EGF-R inhibition drugs in the patents.
»
» Assuming we can obtain this without a prescription, what dosage size in mg
» are we talking about? And also, do we take it orally or incorporate it into
» a topical after the wounding phase? Any ideas?
»
» Edit: I began filling the form out at Goldpharma.com to order 30x 20 mg
» tablets of Arava. Total cost about 152 Euros. But they required to know
» what ailment I would be taking the medication for. I could say arthiritis
» but what if they ask further questions? It seems that they WON’T require a
» prescription from my doctor anyway.
Didn’t look at the side effects of Leflunomide, but if you’re gonna take it internally, why not just crush up and dissolve 1 other pill and use it topically also? This will cover more grounds. Worry if you can do without the other (topically or internal) later on. Both at once can’t be detrimental to one another can it? I just want to hear about you growing a lots of hair:-)
It’s really not a safe assumption that Leflunomide will work through the skin at all. It has to metabolized right and I don’t think anyone knows whether that will happen that way or not.
Orally should be fine if we’re serious about doing this. (Even if it takes 3 days to get the stuff powered-up in the body, there’s still no harm in starting 3 days after wounding instead of 5-6 days, right?)
I’m more worried about trouble with not getting the stuff out than anything. The patents say no more EGF-R inhibition after two weeks from the wounding date. Leflunomide will probably need to be actively flushed out, and even then it may still take a while.
I still like the idea of this stuff better than Gentifib just because overseas Gentifib is such a counterfeit risk. (Absolutely gigantic profit-per-pill ratio, and most users aren’t on it for years & ordering repeated batches.)
And whatever side effects we get from Arava, at least we’re getting a signal that the stuff is legit and the amount in our systems has reached the critical mass. I’d prefer Gentifib if I knew for sure that the source of the drug was 100% legit, but that’s not going to happen for less than several thousand dollars.
If we get this procedure nailed down & growing hair well, then I’ll start thinking of a Gentifib attempt. But right now the only thing I care about is figuring out whether Folica is gonna work AT ALL. F*ck the ideal way to do everything, I just wanna know whether HM is here right now or not for another 15 years. Start trying 10 variables with each attempt and we won’t ever know which one of the 10 isn’t working.
Is it conceivable that taking Arava for just a couple of days will be enough? That way the drug gets into your system and hopefully peters out by the end of the embryonic window (2 weeks after wounding). You know, sort of like a one shot. I’m more than a little concerned about having it in my system after I stop taking it. One reason being that I very much like a drink.
And how do we flush the damned stuff out? I presume that if we only take it for a couple of weeks that it will only take a couple of weeks to exit the body?
Well, either it’s a problem with fading out of the system too fast, or else it’s a problem not being able to get it out of the system when we finally want to. It seems silly to worry about it being BOTH.
Activated charcoal will probably get rid of it. But it might take longer than two days to do it. A few hundred millgrams’ total would probably get expelled in a few weeks one way or the other. But it definitely won’t be gone the first couple of days you stop taking it. I dunno, I might even start trying to expel it a couple of days before the final day we actually intend to inhibit the EGF-R.
Whatever.
Just make sure you don’t start swallowing it until at least day#3 from wounding. Then take a whole lot in the first 2-3 days, and make sure you’re doing something to get rid of it by day #12-14. That’s the most important stuff.
I don’t know what to tell you about alcohol. I don’t know how harmful it really is or isn’t, etc.
Staying off the sauce for a couple of weeks doesn’t bother me personally, so I never thought much about it when I was reading about Leflunomide.
I’ve actually just sent them an email cancelling my order for the drug. To be honest, I have too many concerns regarding it at this time not least my history of excessive alcohol consumption. Good luck with your experiment though. I’m sure if you sprout a phucking big afro< I’ll change my mind and try it.
Well, at least you didn’t bullsh*t yourself about your real chances at doing this clean and then end up hurting yourself with a bad drug interaction. Could be worse.
I don’t know if Gentifib is an alcohol threat or not.
» I still like the idea of this stuff better than Gentifib just because
» overseas Gentifib is such a counterfeit risk.
IMO, it’s a low counterfeit risk. It’s not a popular drug, number one. Number two, no one uses this drug recreationally. Number three, no one who is diagnosed with cancer is going to screw around shopping from some pharmacy in India or some other 3rd-world country via the Internet. This is life or death. They are going to their local pharmacy, and insurance will pay the cost. Those who don’t have insurance can enroll in compassionate care programs set up by drug companies, who will supply the drug for free to those who are dying and can’t afford it.
Also, it would take a sub-human piece of crap to sell fake cancer drugs, as people’s lives are literally riding on the outcome. Almost murder or manslaughter, really. This isn’t like selling pills to get someone’s dick hard.
Also, for the few people who would order gefitinib online, yeah, they could make a few hundred off them, but they would make a hell of a lot more selling fake Viagra, Lipitor, etc. They’d make a lot more from selling fake leflunomide, even. The demand for these drugs would far outstrip the demand for some cancer drug, and they’d wind up making more overall just based on that volume.
» Dermabrasion does not go anywhere near the follicles. It merely affects the
» epidermis and doesn’t go as deep as the dermis, beneath which quite deeply,
» the follicles reside.
baccy, correct me if i’m wrong, but aren’t smaller vellus hairs and especially hair follicles that are in the telogen part of the cycle, much smaller and they don’t extend all the way down into the dermis, they are actually more superficial in the skin?
and if there’s all this dermabrasion going on, repeatedly as benji wants to do, then maybe some of those more superficial smaller follicles will be weakened, and they will loosen and get dislodged or destroyed, with all the constant rubbing over the skin…
i’m not saying dermabrasion will always weaken or destroy normal healthy follicles, but when u are doing this repeated dermabrasion over the same area, then i think that many vulnerable follicles may weaken. eventually some may be just rubbed out of existence by repeated injury.
now if ur talking about guys who already have hairloss and weakened follicles, many of those follicles will be smaller, vellus or in a prolonged telogen phase from mpb. so you’re gonna repeatedly rub at these in hopes of growing more new hair, but what about the ones already there that have been affected by mpb and are kind of miniaturised? they’re more vulnerable and can be destroyed easier.
just my take about why this wounding shyte in its present form isn’t very likely to help us.
» » Dermabrasion does not go anywhere near the follicles. It merely affects
» the
» » epidermis and doesn’t go as deep as the dermis, beneath which quite
» deeply,
» » the follicles reside.
»
» baccy, correct me if i’m wrong, but aren’t smaller vellus hairs and
» especially hair follicles that are in the telogen part of the cycle, much
» smaller and they don’t extend all the way down into the dermis, they are
» actually more superficial in the skin?
»
» and if there’s all this dermabrasion going on, repeatedly as benji wants
» to do, then maybe some of those more superficial smaller follicles will be
» weakened, and they will loosen and get dislodged or destroyed, with all the
» constant rubbing over the skin…
»
» i’m not saying dermabrasion will always weaken or destroy normal healthy
» follicles, but when u are doing this repeated dermabrasion over the same
» area, then i think that many vulnerable follicles may weaken. eventually
» some may be just rubbed out of existence by repeated injury.
»
» now if ur talking about guys who already have hairloss and weakened
» follicles, many of those follicles will be smaller, vellus or in a
» prolonged telogen phase from mpb. so you’re gonna repeatedly rub at these
» in hopes of growing more new hair, but what about the ones already there
» that have been affected by mpb and are kind of miniaturised? they’re more
» vulnerable and can be destroyed easier.
»
» just my take about why this wounding shyte in its present form isn’t very
» likely to help us.
The idea that selling fake Gentifib would be cruel & inhuman is probably not gonna make the counterfeiters think twice.
Anyway, I just remember reading a few articles about fake drugs a while back. I remember one of them specifically mentioned Gentifib among a sentence of several drugs they had found conterfeited during a bust somewhere.
Propecia & Viagra are both known to be BIG in the fake drugs industry. The embarrassment factor + the high price makes it an ideal thing to fake.
In the big picture I know you guys are probably getting real Gentifib. But I just want to know if this Folica deal works so badly.
To be perfectly blunt, the success of this first round of EGF-R tries is probably the difference between me having great hair at 30 years old or still looking like sh*t at 45+ years old. I don’t want to psychologically torture myself by getting no growth and also no side effects from an EGF-R inhibitor, and then spend months wondering whether my drugs were real or not.
Arava is a cheap & safe & available drug. And at least it will probably mess me up enough to know that it was real.
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